nlwxpearo
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 Posted: Thu 15:37, 03 Mar 2011 Post subject: Multi-drug resistant tuberculosis older clinical o |
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,[link widoczny dla zalogowanych]
Multi-drug resistant tuberculosis older clinical observation
Detected on the body, improve accuracy of diagnosis. The current study found that more MP infection can increase airway hyperresponsiveness (AHR), and asthma are closely related, is induced by the potential threat of bronchial asthma ... 3. The possible mechanism of asthma is: (1) MP direct damage: MP by virtue of their unique biological structure of the surface of parasites in the respiratory mucosa. The release of hydrogen peroxide, nucleic acid enzymes substance,[link widoczny dla zalogowanych], swelling of the epithelial cell necrosis and shedding, and cell infiltration and bronchial wall thickening accompanied by small,[link widoczny dla zalogowanych], lumen and affect lung clearance; MP's own biological characteristics which determine AHR histological basis, its continued lurking in the formation of chronic inflammatory airway epithelial stimulation; (2) to promote the release of cytokines and inflammatory mediators: the infection after MP, MP can be settled in airway epithelial cells reproduce, MP can be used as super-antibodies, so that lymphocytes, monocytes, macrophages and other inflammatory cell infiltration, cytokine release 【; (3) to promote growth factor release 【cited, and ultimately can cause chronic inflammation and airway AHR persists, and may further evolve into bronchial asthma. MP preferred erythromycin treatment for pneumonia, and its clear and repeated infection in lung epithelial cells reproduce latent Mycoplasma pneumoniae, Mycoplasma pneumoniae cut off and the whole body caused by persistent pulmonary inflammation results in the affirmative, but the continuing mycoplasma infections hyperresponsiveness can not be alleviated within a short time, resulting in macrolide treatment seems good. In this study, 12 children were accompanied by wheezing, 15.8%; 1 case of lobar pneumonia in children treated in the chest after pulmonary shadows improved increase, change plus azithromycin oral suspension smoking Rute Bu He Lin fluid and budesonide inhalation suspension chest X-ray shadow of the dissipation of 1 week; there were 32 cases of children one to the poor efficacy of erythromycin, given inhaled vasodilator airway symptoms improved, above all show that the mycoplasma infection of airway high response exists, it is in the treatment of mycoplasma pneumonia,[link widoczny dla zalogowanych], such as poor efficacy of erythromycin, do not blindly extend the erythromycin treatment, may be appropriate to increase airway hyperresponsiveness by reducing drug, clinical efficacy. MP pneumonia,[link widoczny dla zalogowanych], and more that the previous light condition, the self-limiting disease, and more without treatment, this view has now been discarded by most scholars. MP severity of clinical symptoms of pneumonia vary, the light only low heat Qing Ke, and sustained high fever accompanied by severe multi-system damage. Most scholars believe that the reason of Mycoplasma antigens and human heart, lung, liver, brain, kidney, smooth muscle there is some common antigens, the infection can produce its own antibodies, causing extrapulmonary manifestations of Mycoplasma pneumonia. Has been reported in patients with pneumonia are 32% MP can be complicated with extrapulmonary manifestations L6J. The MP pneumonia in hospital the past two years, only 1 case of extrapulmonary manifestations, MP pneumonia may be related to early diagnosis and early treatment.
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